About the Book
Very low-carbohydrate (aka ketogenic) diets such as The Atkins Diet, Protein Power and The South Beach Diet have come and gone repeatedly over the years and there is currently great research and real-world interest in their effects. Unfortunately, altogether too much misinformation exists regarding them.
Folks who are pro-low-carbohydrate diets tend to present them as the quick and easy solution to everything including obesity. Easy weight loss without hunger or calorie counting is promised but never seems to pan out as well as we might hope.
At the other extreme are the anti-low-carbohydrate folks who tend to present low-carbohydrate diets as nothing short of a nutritional disaster being perpetrated by a bunch of con men.
The truth, of course lies somewhere in the middle. While low-carbohydrate diets aren’t for everyone and have their pros and cons, the research is clear: they have major benefits under certain circumstances and can be as healthy (and sometimes healthier) than ’standard’ carbohydrate based dieting.
The Ketogenic Diet is the first and only book to objectively examine in-depth the scientific evidence regarding low-carbohydrate/ketogenic diets. It is meant to be a reference manual for low-carbohydrate diets; it is unlike any other book on low-carbohydrate diets that you have ever read or seen.
Covering every topic in extreme detail, The Ketogenic Diet addresses everything from the basic physiology of how the body adapts to a low-carbohydrate intake, the details of human fuel utilization, the impact of low-carbohydrate diets on body composition and many, many more.
Of course, none of the above is useful without practical application guidelines. Details on how to optimize low-carbohydrate diets for different goals (such as fat loss, bodybuilding and endurance performance) are discussed along with three distinct types of low-carbohydrate diets. In addition, the book includes a complete discussion of resistance, aerobic and anaerobic exercise physiology along with specific training programs for different goals and different levels of trainee
At 325 pages and containing over 600 scientific references, this will be your complete reference for ketogenic diets.
Please note: this book does not include information on the ketogenic diet for adolescent epilepsy (the topic is discussed briefly). I highly suggest The Ketogenic Diet: A Treatment for Epilepsy, 3rd Edition (Paperback) by Freeman, Freeman and Kelly (link will take you to Amazon.com page).
Table of contents
Section I: Introduction
1. Introduction to the ketogenic diet
2. History of the ketogenic diet
Section II: The physiology of ketosis
3. Fuel utilization
4. Basic ketone body physiology
5. Adaptations to ketosis
6. Changes in body composition
7. Other effects of the ketogenic diet
Section III: The diets
8. Setting calorie levels
9. The standard ketogenic diet (SKD)
10. Carbs and the ketogenic diet
11. The targeted ketogenic diet (TKD)
12. The cyclical ketogenic diet (CKD)
Section IV: Other topics for the ketogenic diet
13. Breaking fat loss plateaus
14. Ending a ketogenic diet
15. Tools for the ketogenic diet
16. Final considerations
Section V: Exercise physiology
17. Muscular physiology and energy production
18. Aerobic exercise
19. Interval training
20. Weight training
21. The effect of exercise on ketosis
22. Exercise and fat loss
Section VI: Exercise guidelines
23. General exercise guidelines
24. Aerobic exercise
25. Interval training
26. Weight training
Section VII: Exercise programs
27. Beginner programs
28. Intermediate programs
29. The advanced CKD workout
30. Fat loss for pre-competition bodybuilders
Section VIII: Supplements
31. Supplements
Index
Has anyone tried this diet?. Or is it just a pile junk like the rest of his stuff?.
Has anyone tried this diet?. Or is it just a pile junk like the rest of his stuff?.
LOLOLOLOLOLOLOLOLOLOLOLOLOLOLOL
Has anyone tried this diet?. Or is it just a pile junk like the rest of his stuff?.
LOLOLOLOLOLOLOLOLOLOLOLOLOLOLOL
x2
Has anyone tried this diet?. Or is it just a pile junk like the rest of his stuff?.
LOLOLOLOLOLOLOLOLOLOLOLOLOLOLOL
x2
x3 Lyle's a an asshole. But he's an informed and competent asshole with a clear writing style.
x3 Lyle's a an asshole.
his stuff on diet and nutrition is consistently better than anything else on the interwebs.
Calories in, calories out.
Its not rocket science, it's the most simple thing on earth.
"At 325 pages and containing over 600 scientific references", lol.
He's just blagging peoples heads with irrelevant info.
Calories in, calories out.
Its not rocket science, it's the most simple thing on earth.
"At 325 pages and containing over 600 scientific references", lol.
He's just blagging peoples heads with irrelevant info.
Calories in, calories out.
Its not rocket science, it's the most simple thing on earth.
"At 325 pages and containing over 600 scientific references", lol.
He's just blagging peoples heads with irrelevant info.
x3 Lyle's a an asshole.
+4.Quote from: tychverhis stuff on diet and nutrition is consistently better than anything else on the interwebs.
lol.
x3 Lyle's a an asshole.
+4.
lol.
Lyle's a douchebag
then why are you posting/talking about scientific research related to training?
I became interested in the ketogenic diet two and one-half years ago when I used a modified form (called a cyclical ketogenic diet) to reach a level of leanness that was previously impossible using other diets.
Not even past 4th line & he's already talking high.QuoteI became interested in the ketogenic diet two and one-half years ago when I used a modified form (called a cyclical ketogenic diet) to reach a level of leanness that was previously impossible using other diets.
I guess the ketogenic diet is the only diet you can get lean on.
Sillies. You can get lean on a diet of junk food/exercise if you take note of caloric intake.
wow, go write a book and enlighten us all!
the more extreme you want your results to be (according to your body/genetics) the more the "keep it healthy and basic" does not get the job done..
the more extreme you want your results to be (according to your body/genetics) the more the "keep it healthy and basic" does not get the job done..
the more extreme you want your results to be (according to your body/genetics) the more the "keep it healthy and basic" does not get the job done..
Nonsense.
Go tell that to Bolt on his diet of chicken nuggets LMAO.
Bolt is quite possibly the biggest genetic freak of all time and almost certainly on a permanent diet of hugely effective and undetectable drugs.
Nothing he does is relevant to a normal athlete.
Fail on both accounts.
He had no great prospects as a 100m runner/didn't possess the speed. His squat & bench aren't huge (speed isn't primarily made there). Drug use is speculatory at best & since he still hasn't failed a drugs test, the fact remains he's clean till proven guilty.
Yeah, If he followed the ketogenic diet that would put him in the 9.2's, right?.
lol.
Can you imagine a whiteboy getting close to the record, what the reaction would be in the athletic community, if there are so many that believe Bolt is juiced to the gills. Jesus.
Not even past the 4th line & he's already talking high.QuoteI became interested in the ketogenic diet two and one-half years ago when I used a modified form (called a cyclical ketogenic diet) to reach a level of leanness that was previously impossible using other diets.
I guess the ketogenic diet is the only diet you can get lean on.
Sillies. You can get lean on a diet of junk food/exercise as long as you take note of caloric intake.
way i see it, there are three options here:
1. you've still never read anything lyle has written. pretty much no one on the internet is more of an "it depends" guy than lyle, except maybe alan aragon.
2. you're borderline MR and/or illiterate.
3. you're trolling.
He was running mid-low 10s at 16.
Ketogenic diets generally don't work very well for athletes with a high workload.
Past evidence of top sprinters suggest otherwise.
Especially those who pack on large amounts of muscle and annihilate world records. Bolt is more likely to be doping than Christopher Lemaitre, but Lemaitre is probably not clean either.
1. you've still never read anything lyle has written. pretty much no one on the internet is more of an "it depends" guy than lyle, except maybe alan aragon.
Wow, i didn't expect so much BS from you $ick3nin.vend3tta!
Who here believes Casey Combest was on the juice throughout his time in the 100m?.
If so, what makes you believe that?.
http://www.youtube.com/watch?v=Xh-gP2jc3sA
oh and dude you should go read up a little on dieting.
oh and dude you should go read up a little on dieting.
Maintaining 5-8% BF all year round.
I'm aight.
What height/weight? And are those 5-8% internet-bodyfat or real bodyfat?
What height/weight? And are those 5-8% internet-bodyfat or real bodyfat?
6' 0", around 160 lbs. I use body fat calipers to test.
Building for power.
Put on some muscle and see if you can maintain 5% bf ;)
What height/weight? And are those 5-8% internet-bodyfat or real bodyfat?
6' 0", around 160 lbs. I use body fat calipers to test.
Building for power.
Maintaining 5-8% BF all year round.
6' 0", around 160 lbs. I use body fat calipers to test.
Do you have any links/sources which suggest of inaccuracies?.
Where are you pulling the %'s from?.
If they are saying you are 5-8% year round, that's enough to know they are inaccurate. Pro bodybuilders can't even stay 5% year round on tons of drugs.
If they are saying you are 5-8% year round, that's enough to know they are inaccurate. Pro bodybuilders can't even stay 5% year round on tons of drugs.
Come on man, your comparing my lifestyle to the caloric intake/training of a bodybuilder.
Seriously now.
I guess the drugs can't negate the effects of massive caloric consumption/too small amount of cardio activity.
Do you have any links/sources which suggest of inaccuracies?.
Where are you pulling the %'s from?.
Body fat calipers measure skinfolds to calculate how much subcutaneous fat (fat under the skin) a person has. The examiner then puts the numbers into an equation to predict body density and body fat percentage. The point to remember is... Body fat calipers do not measure your body fat percentage directly. The result? There is a great deal of room for errors to creep in.
i dno, my friend eddie (who was in my dunk vids), was "striated" at all times, just insanely lean, ripped to shreds, at 145-150 lb bodyweight/6'0 in shoes.. pretty sure he was at least 6% at all times.
some people are ridiculously lean.
i dno, my friend eddie (who was in my dunk vids), was "striated" at all times, just insanely lean, ripped to shreds, at 145-150 lb bodyweight/6'0 in shoes.. pretty sure he was at least 6% at all times.
some people are ridiculously lean.
You know how i looked some years ago. Skinfold caliper gave me 3,3% bf, bioimpedance just said "error". I surely wasn't 3,3% bf!
What most people think is 5% is more like 10%bf in reality.
Who here believes Casey Combest was on the juice throughout his time in the 100m?.
If so, what makes you believe that?.
“Even today, I’m not a real heavy lifter; I just go heavy enough to develop the muscles. I don’t go after it like some other guys, mostly the shorter runners [laughs]. I’m not really that kind of sprinter.”
(http://www.bcn2010.org/files/13-16524-imatge/Christophe-Lemaitre-nouvel-espoir-de-l-athletisme-francais.jpg)
I find it interesting that nobody thinks Casey Combest was juiced?.
Would that observation/realisation be based more towards his body type (ectomorph) as opposed to a more endomorph/muscular body type?.
Bolt admits he is still only 60 per cent fit after post-Beijing commitments put his winter training behind schedule but he is working flat out to get back into racing shape.
"I'm actually on a diet now because I'm really trying to catch up with my training," he said. "My coach decided that I should stop eating nuggets for a while."
The day of the record-breaking 100 meter final Bolt said he "didn't have breakfast, woke up at 11, sat around watching TV, then had some chicken nuggets, slept for 2 hours...then went back and got some more nuggets!" The reason for the nuggets? Usain was afraid to eat anything else because he didn't trust the foreign food in China.
The UFC allowed Sonnen to have a testosterone level that was 4-times the legal limit, and 16-times the testosterone levels of the average man.
Remember Rashard Lewis?
(http://www.nba.com/magic/photos/lewis00_300_051810.jpg)
Remember Rashard Lewis?
(http://www.nba.com/magic/photos/lewis00_300_051810.jpg)
DHEA. Not even a PED in real world sense of the word.
I HOPE bolt isn't juiced, but i also don't really think he is juiced.. he could have used ped's during his down period between 400m and 100m, but nothing has been proven.. alot of his teammates get busted but he remains clean.. he'd have to fail something or have some people close to him start throwing out accusations, none of which has happened.
casey combest is a "spring" who is extremely strong.. has nothing to do with his physique.. he just has insane bounce, sure he could have been on juice but no one has come out saying he was, he didn't fail anything, etc.. you can't get that kind of bounce with juice, it's natural ability.
Appearance means nothing. Heaps of skinny looking soccer players have tested positive for various anabolic steroids.
Anabolic steroids other than testosterone are pretty easily detected now and athletes have shifted towards using a cocktail of testosterone, HGH, insulin and thyroid hormones which unfortunately are almost almost undetectable. HGH can so far only be detected in blood which is not tested outside of competition. Athletes can use HGH all off season without having to worry. Testosterone testing is based on a ratio of testosterone to epitestosterone. Take moderate doses and keep yourself under 4:1 and again, no worry with testing. Insulin cannot be tested for at all and testing for t3 and t4 is extremely rudimentary. Sprinters and other more general athletes are also using EPO to improve endurance and recovery.
WADA aren't even particularly coy about how easy it is to work around the testing:
http://articles.latimes.com/2007/dec/15/science/sci-baseball15
Dwain Chambers was taking HGH, testosterone, insulin and T3 while being tested multiple times over more than a year and none of it was detected. That's how useless the testing is.
I guess they use it more for training recovery and strength gains (while training). During the competition they might use some stuff that can't be detected.
(http://img190.imageshack.us/img190/679/aestheticcrew.jpg)
Most, if not all of those guys use keto to cut. Granted, some of them are on gear, they still use keto to get them shredded.
inb4 flamed for pic
I've never done keto myself but I did do carb tapering throughout the day and that worked pretty well for me, wasn't very strict either.
I ate chicken. Am I roided? Should I give up on sports?
I know, no and yes.
Usain Bolt could be clean, he could also be using/have used drugs, until proven otherwise its not fair to him to label him a cheater.
Winstrol is a pretty mainstream drug for sprinters, not alot of weight gain, clears quickly, and very popular in power/speed sports.
(http://img190.imageshack.us/img190/679/aestheticcrew.jpg)
Most, if not all of those guys use keto to cut. Granted, some of them are on gear, they still use keto to get them shredded.
inb4 flamed for pic
I've never done keto myself but I did do carb tapering throughout the day and that worked pretty well for me, wasn't very strict either.
(http://img190.imageshack.us/img190/679/aestheticcrew.jpg)
Most, if not all of those guys use keto to cut. Granted, some of them are on gear, they still use keto to get them shredded.
inb4 flamed for pic
I've never done keto myself but I did do carb tapering throughout the day and that worked pretty well for me, wasn't very strict either.
They are all on steroids; test prop, tren, and clen being their drugs of choice.
I thought winstrol made your tendons brittle?.
Usain Bolt could be clean, he could also be using/have used drugs, until proven otherwise its not fair to him to label him a cheater.
Winstrol is a pretty mainstream drug for sprinters, not alot of weight gain, clears quickly, and very popular in power/speed sports.
i thought winstrol made your tendons brittle? i figured more athletes(who can afford it)would use anavar
Anyway, I dont like talking about steroids period, I dont condone it and I dont believe its necessary in any sport other than bodybuilding.. Ive said this every time this subject comes up, but there are 1000x more frat boys in your local gym JUICED TO THE GILLS, weak as dogshit, and good at absolutely no form of anything resembling athleticism, than there are elite or good athletes on drugs.
Remember Rashard Lewis?
(http://www.nba.com/magic/photos/lewis00_300_051810.jpg)
DHEA. Not even a PED in real world sense of the word.
What are the anabolic steroids, HGH, t3 and t4 improving in terms of sprint speed?. I associate those drugs with building muscle. Combest & Lemaitre don't have big muscles. So, what are those drugs doing to aid the skinny guys?. How/what are they gaining from usage?.
But the thing is, those guys don't even look like they lift. If they both walked down the street towards me I wouldn't associate them with lifting weights (even light weights).
I ate chicken. Am I roided? Should I give up on sports?
I know, no and yes.
Chicken isn't made in laboratories
inb4 dem hormones in chicken
Whenever people ask me about steroids and GH in meat, I'm pointing to this kid: see, the stuff's helping.
I ate chicken. Am I roided? Should I give up on sports?
I know, no and yes.
Chicken isn't made in laboratories
inb4 dem hormones in chicken
Posted Hickson's 800 lb DL video in Lyle's forum. He said this:Quote from: Lyle McDonaldWhenever people ask me about steroids and GH in meat, I'm pointing to this kid: see, the stuff's helping.
I ate chicken. Am I roided? Should I give up on sports?
I know, no and yes.
Chicken isn't made in laboratories
inb4 dem hormones in chicken
Posted Hickson's 800 lb DL video in Lyle's forum. He said this:Quote from: Lyle McDonaldWhenever people ask me about steroids and GH in meat, I'm pointing to this kid: see, the stuff's helping.
Yeah. It's scary enough the Alberto Contador's excuse for clenbuterol was seriously fucking plausable.
philray is natty. He has progress pics of himself since he started lifting... looks natty, no huge jumps. He's also the oldest and has been working out the longest.
Babo claims he's natty. Skeptical though because he blew up just like zyzz/chestbrah in the same time period and they are open about using gear. Babo says his grandpa or something was some successful bodybuilder or something though so he claims he has very good genetics.
Manlets ftw
philray is natty. He has progress pics of himself since he started lifting... looks natty, no huge jumps. He's also the oldest and has been working out the longest.
Babo claims he's natty. Skeptical though because he blew up just like zyzz/chestbrah in the same time period and they are open about using gear. Babo says his grandpa or something was some successful bodybuilder or something though so he claims he has very good genetics.
Manlets ftw
None of them are natty. And all of them "look natty". None of them have impressive physiques that you couldn't get naturally.
philray is natty. He has progress pics of himself since he started lifting... looks natty, no huge jumps. He's also the oldest and has been working out the longest.
Babo claims he's natty. Skeptical though because he blew up just like zyzz/chestbrah in the same time period and they are open about using gear. Babo says his grandpa or something was some successful bodybuilder or something though so he claims he has very good genetics.
Manlets ftw
None of them are natty. And all of them "look natty". None of them have impressive physiques that you couldn't get naturally.
can we have a rule on this forum to never use the term "natty" again? just such a bro douche bag thing to say
for the record, he was joking.
philray is natty. He has progress pics of himself since he started lifting... looks natty, no huge jumps. He's also the oldest and has been working out the longest.
Babo claims he's natty. Skeptical though because he blew up just like zyzz/chestbrah in the same time period and they are open about using gear. Babo says his grandpa or something was some successful bodybuilder or something though so he claims he has very good genetics.
Manlets ftw
None of them are natty. And all of them "look natty". None of them have impressive physiques that you couldn't get naturally.
can we have a rule on this forum to never use the term "natty" again? just such a bro douche bag thing to say
x2
:)
philray is natty. He has progress pics of himself since he started lifting... looks natty, no huge jumps. He's also the oldest and has been working out the longest.
Babo claims he's natty. Skeptical though because he blew up just like zyzz/chestbrah in the same time period and they are open about using gear. Babo says his grandpa or something was some successful bodybuilder or something though so he claims he has very good genetics.
Manlets ftw
None of them are natty. And all of them "look natty". None of them have impressive physiques that you couldn't get naturally.
can we have a rule on this forum to never use the term "natty" again? just such a bro douche bag thing to say
x2
:)
That's because your associating it with a favorite of bros everywhere, natty ice and natty light.
Increased recovery
Increased growth of both muscles and tendons
Reduced body fat
Increased collagen synthesis for healthy joints
Increased strength
Increased injury resistance
Able to handle more volume in a training session
Improved ability to maintain pace
Yeah. It's scary enough the Alberto Contador's excuse for clenbuterol was seriously fucking plausable.
Increased recovery
Increased growth of both muscles and tendons
Reduced body fat
Increased collagen synthesis for healthy joints
Increased strength
Increased injury resistance
Able to handle more volume in a training session
Improved ability to maintain pace
I won't argue all your points but one I want your opinion on the most, increased growth of tendons through steroid use.
Something I just pulled from another board regarding steroids and tendons. What do you make of the article tychver?. One sentence remarks: "winstrol increases collagen synthesis. It will give you bigger tendons. However, your body compensates for this by making them more brittle, weaker, and more prone to injury".
Original Link: http://www.trainwiser.com/f100/steroids-tendons-4839/
Steroids and tendons.
While injecting test increases protein synthesis by roughly 50 times, depending on dose and time, most bodybuilders forget that it will reduce collagen synthesis by more than 50% -- more like 80%, giving you the collagen synthesis rate of a senior citizen. Since collagen makes up tendons, bros are very prone to injury if they continue to lift very heavy, unless they cycle off T and let their collagen synthesis get back to normal. It's like having the skeletal muscle of a gorilla with the tendons of a very old man.
Winstrol increases collagen synthesis. It will give you bigger tendons. However, your body compensates for this by making them more brittle, weaker, and more prone to injury. I can't tell you how many bros work out anaerobically and become injured while on winstrol. Guys who lift in the 1-5 rep range while on winstrol, to baseball players who sprint all out from a stationary position -- winstrol should be the LAST drug they choose. Most of them like winstrol because they don't get the weight gain from it but it is very detrimental to bros who train for any sport anaerobically. Tendons tear easily on it.
Also, the drugs I mention increase collagen synthesis while also increasing collagen cross-linking integrity, making for a much stronger tendon.
Winstrol, on the other hand, will dramatically increase collagen syn, but ironically it decreases collagen cross-linking integrity, thus making a much weaker tendon.
You can plan a cycle of AAS which will increase collagen synthesis and skeletal muscle growth at the same time. The key is the drug(s) you choose.
Deca, Equipoise, Anavar, and Primobolan will ALL increase skeletal muscle while at the same time dramatically increase collagen syn and bone mass and density, leaving you with a substantially reduced chance of becoming injured than if you choose to use AAS like sus, cyp, or enth.
While testosterone will increase bone mass and density, even at supra-physiological levels, the result is weaker tendons due to inhibition of collagen syn.
To plan a cycle where the goal is to increase skeletal muscle mass/strength while at the same time increase joint/tendon/ligament strength, enough to keep up with the dramatic increase in skeletal muscle, you must choose drugs like Eq, Deca, Anavar, or Primo as the base of your cycle. Testosterone and its esters can be added to your cycle to keep levels within a 'normal' physiological range (ie, 100-200 mg/wk) but must not go above this. Since drugs like eq, deca, anavar and primo will reduce endogenous, natural levels of test, these levels may be maintained with exogenous test in the 100-200 mg/wk range. Test at this dose will not inhibit collagen syn, but paradoxically, will help increase it. It is when exogenous testosterone is used > 200 mg/wk that collagen syn is inhibited.
Deca @ 3 mg/kg a week(about 270 mg/wk for a 200 lb male) will increase procollagen III levels by 270% by week 2. Procollagen III is a primary indicator used to determine the rate of collagen syn. As you can see, deca is a very good drug at giving you everything you want -- an increase in collagen syn, an increase in skeletal muscle, and increases in bone mass and density. The one thing it does not give you is wood
Primobolan, @ 5 mg/kg, will increase collagen synthesis by roughly 180% -- less than deca and equipoise but still substantial.
Equipoise @ 3 mg/kg will increase procollagen III by approximately 340% -- slightly better than deca.
Oxandrolone has over a hundred studies documenting its effectiveness at treating patients needing rapid increases in collagen syn to enhance healing.
These drugs have longer half-lives than most other AAS, so this should be considered when timing your post cycle clomid use. Here they are:
Deca: 15 days Equipoise: 14 days Primobolan: 10.5 days
Anavar has a half-life of only 8 hours so it should not pose a problem.
GH is probably the most remarkable drug at increasing collagen synthesis. It increases collagen syn in a dose dependant manner -- the more you use, the more you will increase collagen syn. It has also demonstrated this ability in short and long term studies. From what I've read, hGH at 6 iu/day increased the collagen deposition rate by around 250% in damaged collagen structures. This result indicates that the increased biomechanical strength of wounds to collagen structures treated with biosynthetic human growth hormone was produced by an increased deposition of collagen in the collagen structures.
Eq, primo, anavar, and deca are all good -- they increase several biomakers of collagen syn -- ie, type III, II, I, procollagen markers. GH just seems to do so most dramatically.
Use of any of these drugs @ supra-physiological levels with a maintenance dose of test will increase collagen syn while at the same time increase skeletal muscle mass. Skeletal muscle mass gains will not be as dramatic as with large testosterone doses but you have to weigh the risk/reward basis for yourself. Also, these drugs do not satisfy the libido like testosterone, but that is not the point of this thread. It is only to demonstrate that you can increase skeletal muscle and collagen syn at the same time with certain AAS, the decision is up to you.Yeah. It's scary enough the Alberto Contador's excuse for clenbuterol was seriously fucking plausable.
+1.
Pharmador should have received a 2 year ban. I'm a huge cycling fan & the UCI is corrupt from top to bottom. It's a complete joke.
(http://www.bikerumor.com/wp-content/uploads/2010/09/contador-on-a-cow.jpg)
philray is natty. He has progress pics of himself since he started lifting... looks natty, no huge jumps. He's also the oldest and has been working out the longest.
Babo claims he's natty. Skeptical though because he blew up just like zyzz/chestbrah in the same time period and they are open about using gear. Babo says his grandpa or something was some successful bodybuilder or something though so he claims he has very good genetics.
Manlets ftw
None of them are natty. And all of them "look natty". None of them have impressive physiques that you couldn't get naturally.
can we have a rule on this forum to never use the term "natty" again? just such a bro douche bag thing to say
x2
:)
When It Comes to Building Tendons, Not All Steroids are Created Equal
by Hypertrophy
CEM-Meso.com
Stimulation of collagen synthesis by the anabolic steroid stanozolol.
Researchers: Falanga V, Greenberg AS, Zhou L, Ochoa SM, Roberts AB, Falabella A, Yamaguchi Y; University of Miami School of Medicine, Department of Dermatology, Miami, Veterans Affairs Medical Center, Florida, USA.
Source: J Invest Dermatol 1998 Dec;111(6):1193-7
Summary: In this report, we measured the effect of the anabolic steroid stanozolol on cell replication and collagen synthesis in cultures of adult human dermal fibroblasts. Stanozolol (0.625-5 micrograms per ml) had no effect on fibroblast replication and cell viability but enhanced collagen synthesis in a dose-dependent manner. Stanozolol also increased (by 2-fold) the mRNA levels of alpha1 (I) and alpha1 (III) procollagen and, to a similar extent, upregulated transforming growth factor-beta1 (TGF-beta1) mRNA and peptide levels. There was no stimulation of collagen synthesis by testosterone. The stimulatory effects of stanozolol on collagen synthesis were blocked by a TGF-beta1 anti-sense oligonucleotide, by antibodies to TGF-beta, and in dermal fibroblast cultures derived from TGF-beta-1 knockout mice. We conclude that collagen synthesis is increased by the anabolic steroid stanozolol and that, for the most part, this effect is due to TGF-beta-1. These findings point to a novel mechanism of action of anabolic steroids.
Discussion: I must first acknowledge that the commonly held belief is that anabolic steroids predispose an athlete to tendon rupture. This conclusion is drawn from animal studies showing that some steroids produce a larger, stiffer tendon in rats and that these steroid-induced tendons "fail" before the tendons from the control animals. The term fail refers to the breaking point.
The interesting thing about the present study is that the steroid stanozolol (Winstrol) had a different effect than testosterone. If you are a regular reader of MESO-Rx you should be well aware that not all steroids act in the same manner. And that because of subtle differences in there molecular structure they are able to elicit different responses. For example, Deca seems to act primarily through the androgen receptor (AR) where as Dianabol has effects beyond those associated with the AR.
Because synthetic steroids have differ in their chemical properties it should not be surprising that testosterone did not have the same effect as Winstrol. Winstrol increased collagen synthesis as opposed to testosterone which did not in this study. Interpreting the results of this study are more difficult than simply describing them. Other researchers have suggested that steroids cause a rapid increase in protein synthesis within tendon fibroblasts which results in fibroids or fibrous nodules within the tendon (Michna,1988). These fibroids alter the mechanical properties of the tendon perhaps predisposing it to rupture. It is also noted that during short term use of steroids there is an alteration in the alignment of collagen fibers which may also lead to rupture. Interestingly these alterations in collagen metabolism are transient with markers of collagen turnover returning more or less to baseline after 3-4 weeks of steroid administration (Karpakka,1992). These same researchers noted that low dose anabolics effect primarily muscle collagenous tissue with tendon being effected only at higher doses (i.e. 5 times the therapeutic dose) which would more closely represent what is needed by bodybuilders to put on mass.
The question remains, dose this mean that Winstrol will actually help prevent tendon injury or will it lead to bigger yet stiffer tendons prone to injury? It is difficult to take animal research and extrapolate the results to humans. Stanozolol is used therapeutically in humans to treat a variety of connective tissue and vascular disorders and its clinical effects suggest that it can modulate connective tissue breakdown in people. Despite being labeled as "ineffective" by many bodybuilders it is very popular among athletes. As with most hormones, dosage plays a role in what effects are seen, be they positive or negative. Hopefully future studies will shed light on the therapeutic effects of different steroids on tendons in humans.
References:
Michna H Appearance and ultrastructure of intranuclear crystalloids in tendon fibroblasts induced by an anabolic steroid hormone in the mouse. Acta Anat (Basel) 1988;133(3):247-50
Karpakka JA, Pesola MK, Takala TE. The effects of anabolic steroids on collagen synthesis in rat skeletal muscle and tendon. A preliminary re
Read more from this MESO-Rx article at: http://www.mesomorphosis.com/articles/haycock/anabolic-steroids-and-collagen-synthesis.htm#ixzz1IDM8nfTZ
When It Comes to Building Tendons, Not All Steroids are Created Equal
by Hypertrophy
CEM-Meso.com
Stimulation of collagen synthesis by the anabolic steroid stanozolol.
Researchers: Falanga V, Greenberg AS, Zhou L, Ochoa SM, Roberts AB, Falabella A, Yamaguchi Y; University of Miami School of Medicine, Department of Dermatology, Miami, Veterans Affairs Medical Center, Florida, USA.
Source: J Invest Dermatol 1998 Dec;111(6):1193-7
Summary: In this report, we measured the effect of the anabolic steroid stanozolol on cell replication and collagen synthesis in cultures of adult human dermal fibroblasts. Stanozolol (0.625-5 micrograms per ml) had no effect on fibroblast replication and cell viability but enhanced collagen synthesis in a dose-dependent manner. Stanozolol also increased (by 2-fold) the mRNA levels of alpha1 (I) and alpha1 (III) procollagen and, to a similar extent, upregulated transforming growth factor-beta1 (TGF-beta1) mRNA and peptide levels. There was no stimulation of collagen synthesis by testosterone. The stimulatory effects of stanozolol on collagen synthesis were blocked by a TGF-beta1 anti-sense oligonucleotide, by antibodies to TGF-beta, and in dermal fibroblast cultures derived from TGF-beta-1 knockout mice. We conclude that collagen synthesis is increased by the anabolic steroid stanozolol and that, for the most part, this effect is due to TGF-beta-1. These findings point to a novel mechanism of action of anabolic steroids.
Discussion: I must first acknowledge that the commonly held belief is that anabolic steroids predispose an athlete to tendon rupture. This conclusion is drawn from animal studies showing that some steroids produce a larger, stiffer tendon in rats and that these steroid-induced tendons "fail" before the tendons from the control animals. The term fail refers to the breaking point.
The interesting thing about the present study is that the steroid stanozolol (Winstrol) had a different effect than testosterone. If you are a regular reader of MESO-Rx you should be well aware that not all steroids act in the same manner. And that because of subtle differences in there molecular structure they are able to elicit different responses. For example, Deca seems to act primarily through the androgen receptor (AR) where as Dianabol has effects beyond those associated with the AR.
Because synthetic steroids have differ in their chemical properties it should not be surprising that testosterone did not have the same effect as Winstrol. Winstrol increased collagen synthesis as opposed to testosterone which did not in this study. Interpreting the results of this study are more difficult than simply describing them. Other researchers have suggested that steroids cause a rapid increase in protein synthesis within tendon fibroblasts which results in fibroids or fibrous nodules within the tendon (Michna,1988). These fibroids alter the mechanical properties of the tendon perhaps predisposing it to rupture. It is also noted that during short term use of steroids there is an alteration in the alignment of collagen fibers which may also lead to rupture. Interestingly these alterations in collagen metabolism are transient with markers of collagen turnover returning more or less to baseline after 3-4 weeks of steroid administration (Karpakka,1992). These same researchers noted that low dose anabolics effect primarily muscle collagenous tissue with tendon being effected only at higher doses (i.e. 5 times the therapeutic dose) which would more closely represent what is needed by bodybuilders to put on mass.
The question remains, dose this mean that Winstrol will actually help prevent tendon injury or will it lead to bigger yet stiffer tendons prone to injury? It is difficult to take animal research and extrapolate the results to humans. Stanozolol is used therapeutically in humans to treat a variety of connective tissue and vascular disorders and its clinical effects suggest that it can modulate connective tissue breakdown in people. Despite being labeled as "ineffective" by many bodybuilders it is very popular among athletes. As with most hormones, dosage plays a role in what effects are seen, be they positive or negative. Hopefully future studies will shed light on the therapeutic effects of different steroids on tendons in humans.
References:
Michna H Appearance and ultrastructure of intranuclear crystalloids in tendon fibroblasts induced by an anabolic steroid hormone in the mouse. Acta Anat (Basel) 1988;133(3):247-50
Karpakka JA, Pesola MK, Takala TE. The effects of anabolic steroids on collagen synthesis in rat skeletal muscle and tendon. A preliminary re
Read more from this MESO-Rx article at: http://www.mesomorphosis.com/articles/haycock/anabolic-steroids-and-collagen-synthesis.htm#ixzz1IDM8nfTZ
so basically this study says it doesn't know
When It Comes to Building Tendons, Not All Steroids are Created Equal
by Hypertrophy
CEM-Meso.com
Stimulation of collagen synthesis by the anabolic steroid stanozolol.
Researchers: Falanga V, Greenberg AS, Zhou L, Ochoa SM, Roberts AB, Falabella A, Yamaguchi Y; University of Miami School of Medicine, Department of Dermatology, Miami, Veterans Affairs Medical Center, Florida, USA.
Source: J Invest Dermatol 1998 Dec;111(6):1193-7
Summary: In this report, we measured the effect of the anabolic steroid stanozolol on cell replication and collagen synthesis in cultures of adult human dermal fibroblasts. Stanozolol (0.625-5 micrograms per ml) had no effect on fibroblast replication and cell viability but enhanced collagen synthesis in a dose-dependent manner. Stanozolol also increased (by 2-fold) the mRNA levels of alpha1 (I) and alpha1 (III) procollagen and, to a similar extent, upregulated transforming growth factor-beta1 (TGF-beta1) mRNA and peptide levels. There was no stimulation of collagen synthesis by testosterone. The stimulatory effects of stanozolol on collagen synthesis were blocked by a TGF-beta1 anti-sense oligonucleotide, by antibodies to TGF-beta, and in dermal fibroblast cultures derived from TGF-beta-1 knockout mice. We conclude that collagen synthesis is increased by the anabolic steroid stanozolol and that, for the most part, this effect is due to TGF-beta-1. These findings point to a novel mechanism of action of anabolic steroids.
Discussion: I must first acknowledge that the commonly held belief is that anabolic steroids predispose an athlete to tendon rupture. This conclusion is drawn from animal studies showing that some steroids produce a larger, stiffer tendon in rats and that these steroid-induced tendons "fail" before the tendons from the control animals. The term fail refers to the breaking point.
The interesting thing about the present study is that the steroid stanozolol (Winstrol) had a different effect than testosterone. If you are a regular reader of MESO-Rx you should be well aware that not all steroids act in the same manner. And that because of subtle differences in there molecular structure they are able to elicit different responses. For example, Deca seems to act primarily through the androgen receptor (AR) where as Dianabol has effects beyond those associated with the AR.
Because synthetic steroids have differ in their chemical properties it should not be surprising that testosterone did not have the same effect as Winstrol. Winstrol increased collagen synthesis as opposed to testosterone which did not in this study. Interpreting the results of this study are more difficult than simply describing them. Other researchers have suggested that steroids cause a rapid increase in protein synthesis within tendon fibroblasts which results in fibroids or fibrous nodules within the tendon (Michna,1988). These fibroids alter the mechanical properties of the tendon perhaps predisposing it to rupture. It is also noted that during short term use of steroids there is an alteration in the alignment of collagen fibers which may also lead to rupture. Interestingly these alterations in collagen metabolism are transient with markers of collagen turnover returning more or less to baseline after 3-4 weeks of steroid administration (Karpakka,1992). These same researchers noted that low dose anabolics effect primarily muscle collagenous tissue with tendon being effected only at higher doses (i.e. 5 times the therapeutic dose) which would more closely represent what is needed by bodybuilders to put on mass.
The question remains, dose this mean that Winstrol will actually help prevent tendon injury or will it lead to bigger yet stiffer tendons prone to injury? It is difficult to take animal research and extrapolate the results to humans. Stanozolol is used therapeutically in humans to treat a variety of connective tissue and vascular disorders and its clinical effects suggest that it can modulate connective tissue breakdown in people. Despite being labeled as "ineffective" by many bodybuilders it is very popular among athletes. As with most hormones, dosage plays a role in what effects are seen, be they positive or negative. Hopefully future studies will shed light on the therapeutic effects of different steroids on tendons in humans.
References:
Michna H Appearance and ultrastructure of intranuclear crystalloids in tendon fibroblasts induced by an anabolic steroid hormone in the mouse. Acta Anat (Basel) 1988;133(3):247-50
Karpakka JA, Pesola MK, Takala TE. The effects of anabolic steroids on collagen synthesis in rat skeletal muscle and tendon. A preliminary re
Read more from this MESO-Rx article at: http://www.mesomorphosis.com/articles/haycock/anabolic-steroids-and-collagen-synthesis.htm#ixzz1IDM8nfTZ
so basically this study says it doesn't know
basically, that study shows that winstrol is used to IMPROVE connective tissue in certain cases and you cant draw human conclusions based on rat research. So basically, its just showing how the idiots who go around saying "winstrol causes tendon damage" got their flawed info from, basically.
Having just done a google search, I'm hearing so many stories of tendon ruptures following steroid usage?.
basically, that study shows that winstrol is used to IMPROVE connective tissue in certain cases and you cant draw human conclusions based on rat research. So basically, its just showing how the idiots who go around saying "winstrol causes tendon damage" got their flawed info from, basically.
so basically this study says it doesn't know.
When you combine that with the fact that they've often never lifted properly before and their bodies are not well adapted to the stress of repeatedly lifting heavy you have a disaster waiting to happen. But you wanna look jacked for the bitchez bro.
Weyand and his associates proved that simply gaining strength is not enough. Their study showed that the key to faster running was mass-specific force. ‘Mass-specific force’ is just another way to say that it isn’t merely the amount of force applied to the ground that increases stride length; it’s the amount of force in relation to bodyweight.
Let’s look at an example to clear up any possible confusion:
We have two miniature rockets, A and B, that are of equal size, carry equal amounts of fuel, and use engines of equal power. The only difference between A and B is their weight. A is made out of heavy steel and weighs in at a hefty 100 pounds but B is made of lightweight material that weighs only 50 pounds.
The engines fire.
What happens? B blows off its launch pad before A, quickly puts an increasing amount of distance between them, then watches as A’s added weight causes it to drain its fuel supply and drop like a brick.
All other things being equal, the lighter rocket will go faster and further every time.
basically, that study shows that winstrol is used to IMPROVE connective tissue in certain cases and you cant draw human conclusions based on rat research. So basically, its just showing how the idiots who go around saying "winstrol causes tendon damage" got their flawed info from, basically.
Bro science? In bodybuilding?
For the people who genuinely believe most sprinters are juiced, is there anything in there persona/attitude/build etc that may convince you otherwise?.
Can't really believe a word anyone says.
Can't really believe a word anyone says.
Would that also apply to you JC if you was a professional athlete?.
For the people who genuinely believe most sprinters are juiced, is there anything in there persona/attitude/build etc that may convince you otherwise?.
For the people who genuinely believe most sprinters are juiced, is there anything in there persona/attitude/build etc that may convince you otherwise?.
i don't just assume everyone is juicing.. when people fail tests though it's over for me respecting them.. so if usain failed one, adios.. but until then he's clean, so is tyson gay etc.
It takes ridiculous genetics to run a sub 10, even with steroids.