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Electromyostimulation as part of a short strength-training program enhanced knee extensor strength and squat jump performance of basketball players

These results showed that an elcctrostimulation program of the latissimus dorsi increased the strength and swimming performances of a group of competitive swimmers.

Group 1 trained with maximally tolerable isometric contractions induced by ES (ELECTRICAL STIMULATION), three days a week for four weeks. Results showed that although both groups demonstrated increases in isometric strength of their quadriceps femoris muscles, training isometrically with ES produced a significantly greater increase (p < .01) than not training with ES.

The patients who received neuromuscular electrical stimulation had stronger quadriceps muscles and more normal gait patterns than those in the volitional exercise group.

In sports medicine, neuromuscular electrical stimulation (NMES) has been used for muscle strengthening, maintenance of muscle mass and strength during prolonged periods of immobilisation, selective muscle retraining, and the control of oedema. A wide variety of stimulators, including the burst-modulated alternating current ('Russian stimulator'), twin-spiked monophasic pulsed current and biphasic pulsed current stimulators, have been used to produce these effects. Several investigators have reported increased isometric muscle strength in both NMES-stimulated and exercise-trained healthy, young adults when compared to unexercised controls, and also no significant differences between the NMES and voluntary exercise groups. It appears that when NMES and voluntary exercise are combined there is no significant difference in muscle strength after training when compared to either NMES or voluntary exercise alone. There is also evidence that NMES can improve functional performance in a variety of strength tasks. Two mechanisms have been suggested to explain the training effects seen with NMES. The first mechanism proposes that augmentation of muscle strength with NMES occurs in a similar manner to augmentation of muscle strength with voluntary exercise. This mechanism would require NMES strengthening protocols to follow standard strengthening protocols which call for a low number of repetitions with high external loads and a high intensity of muscle contraction. The second mechanism proposes that the muscle strengthening seen following NMES training results from a reversal of voluntary recruitment order with a selective augmentation of type II muscle fibres. Because type II fibres have a higher specific force than type I fibres, selective augmentation of type II muscle fibres will increase the overall strength of the muscle. The use of neuromuscular electrical stimulation to prevent muscle atrophy associated with prolonged knee immobilisation following ligament reconstruction surgery or injury has been extensively studied. NMES has been shown to be effective in preventing the decreases in muscle strength, muscle mass and the oxidative capacity of thigh muscles following knee immobilisation. In all but one of the studies, NMES was shown to be superior in preventing the atrophic changes of knee immobilisation when compared to no exercise, isometric exercise of the quadriceps femoris muscle group, isometric co-contraction of both the hamstrings and quadriceps femoris muscle groups, and combined NMES-isometric exercise. It has also been reported that NMES applied to the thigh musculature during knee immobilisation improves the performance on functional tasks.

Such stimulation should be a valuable modality for developing isometric strength when normal voluntary motion is hampered. However, it appears to have little applicability to developing the kind of strength associated with rapid movements.

In conclusion glycolysis produced approximately 195 mmol ATP/kg dry muscle during the initial 48 contractions (76.8 s) and only approximately 15 mmol ATP/kg dry muscle during the final 16 contractions. Equivalent values for total ATP turnover were 278 and 16.5 mmol/kg dry muscle.

We concluded that the voluntary torque losses observed after detraining could be attributed to both neural and muscular alterations. Muscle size preservation could explain the higher knee extensor MVC values observed after the cessation of training compared to those obtained before training, therefore indicating that muscle size changes are slower than neural drive reduction.

Conclusion: EMS combined with plyometric training has proven useful for the improvement of vertical jump ability in volleyball players. This combined training modality produced rapid increases (~2 wk) of the knee extensors and plantar flexors maximal strength. These adaptations were then followed by an improvement in general and specific jumping ability, likely to affect performance on the court. In conclusion, when EMS resistance training is proposed for vertical jump development, specific work out (e.g., plyometric) must complement EMS sessions to obtain beneficial effects.

Adarqui,
  My name is Dave Brewer. I am a coach  in Orange County, California. I have followed your youtube site for over a year. Absolutely great stuff. I work with Football, Girls Soccer, Girls Basketball, baseball, rowing, and some throwers. I was a competitive Powerlifter, and some Olympic weightlifting. This area is a hot bed for athletes and standard/cookie cutter training programs. I am in the process of setting up a small training facility. Even with the economy, I see the potential. Hope your site goes well!!

My only goal in life is to dunk.

Quote from: Joe on June 05, 2009, 03:56:08 am

um, wtf?

I thought the same thing until I realized that even if she has a vertical of around 16 inches, mine is only five inches higher and I'm definitely older and bigger than her.

I can't believe you put up CCJ's "forumula"...